Hinz Medical Foods Logo

For management of symptoms due to
hyposerotonergicTM, hypodopaminergicTM, and hypoglutathionemiaTM conditions
on the “optimally modified normal diet”.TM

For optimal medical food results, orientation and training are required

Six-hours online teaching conferences
(Two three-hour live days)

2-Day Course to select from 2 Series

Series 1 – Saturday, Sept. 17 and Saturday, Sept. 24, 2022
Series 2 – Saturday, Oct. 15 and Saturday, Oct. 22, 2022

Conferences are from 9 am to noon central time.

This research is not static. It continues to grow and move forward. For more information or to register click here.

Hinz Medical Foods
(Pills or Tablets)

    Hinz Medical Foods are for the management of symptoms caused by low serotonin, low dopamine, or low glutathione which may accompany and be identical to disease symptoms.

    Medical foods are not nutritional supplements. FDA regulations required the administration of medical foods for the management of nutritional conditions such as these. Under FDA regulations nutritional supplements are not allowed to make treatment claims for these conditions.

    When symptoms caused by low serotonin, low dopamine, and low glutathione exist, failing to provide the necessary amino acids will do nothing to address the cause of the problem and nothing to increase the synthesis of serotonin, dopamine, or glutathione.

    Drugs that work with neurotransmitters do not work if there are not enough neurotransmitters. The only way to increase the synthesis of the total number of serotonin or dopamine molecules synthesized in the central nervous system is by administering the required nutrients.

This Course Teaches

Management of disease (listed on the right) symptoms present on an optimally modified diet which may be accompanied by and identical to symptoms caused by:

  • low serotonin (hyposerotonergic™ condition)
  • low dopamine (hypodopaminergic™ condition)
  • low glutathione (hypoglutathionemia™ condition

When symptoms are caused by low serotonin, dopamine, or glutathione, not providing the necessary nutrients, including amino acids, will do nothing to address the cause of the problem and the need to increase the synthesis of serotonin, dopamine, and glutathione.

MIGRAINE AND THE SEROTONIN AGONIST

    PRESCRIBING INFORMATION: “Sumatriptan (Imitrex®) presumably exerts its therapeutic effects in treating migraine headaches through agonist effects at the 5-HT1B/1D receptors on intracranial blood vessels.”

    Consider migraine headaches. Sumatriptan (Imitrex®) exerts its serotonin agonist effects on the 5-HT1B/D (serotonin) receptors. The hyposerotonergic™ condition is where serotonin concentrations are too low to affect the required activation of the 5-HT1B/D receptors on the optimally modified normal diet. When a drug functions as an agonist, it is essentially fake serotonin stimulating the receptors. Increasing serotonin synthesis requires administering the nutrients the body requires for the relief of symptoms. When the patient is diagnosed with migraine headache and managing a hyposerotonergic condition with nutrients relieves all symptoms, the cause of the headaches is not migraine. It is a hyposerotonergic condition (low serotonin).

This course teaches the ability to manage symptoms caused by low serotonin, dopamine, or glutathione which may be identical to symptoms of diseases listed to the right.

  • The course teaches the ability to differentiate symptoms associated with a disease, such as low serotonin or dopamine.

FAT-SOLUBLE TOXIN MANAGEMENT

  • Optimal management of glutathione inside and outside of the brain requires L-cysteine.
  • L-cysteine is the rate-limiting step in glutathione synthesis. Lcysteine crosses the blood-brain barrier; Glutathione and NAC do not cross the blood-brain barrier.
  • Glutathione is the body’s most potent agent for neutralizing fat-soluble toxins.
  • In humans, we have developed firsthand data demonstrating the optimal daily dose of L-cysteine.
  • This course teaches how the primary problem with administering glutathione and NAC is not prescribing enough, and they do not cross the blood-brain barrier.
  • Chronic disease is commonly associated with collapsed (depleted) glutathione.
  • Collapse (depletion) of glutathione with all chronic diseases and aging is typical.

 

Day 1: General Protocols

When symptoms associated with diseases below are caused by low serotonin, dopamine, or glutathione, not providing the necessary nutrients, including amino acids, will do nothing to address the cause of the problem.

    When the differential diagnosis determines whether any of the following diseases are present, a secondary differential diagnosis is require to determine if a hyposerotonergic™, hypodopaminergic™, or hypoglutahionemia™ condition is present. The following is a list of conditions where secondary issues requiring medical food for these conditions may be present.

Start hypodopaminergic protocol for hypodopaminergic conditions

Parkinson’s disease
Restless Leg Syndrome

Start the hyposerotonergic / hypodopaminergic protocol

Addiction
Alzheimer’s (dementia)
ADHD / ADD
Adrenal fatigue
Alcoholism
Allergies (histamine)
    Allergy induced asthma
    Chemical sensitivities
    Peanut or other foods
    Urticaria
Anxiety
Autism
Bipolar
Monoamine depletion:
    Chronic illness
    Chronic pain
    Chronic stress
Cognitive deterioration
Depression
Eating Disorders
Essential tremor
    Rule out Parkinson’s disease
Fatigue (Negative metabolic workup)
Fibromyalgia

GABA dysfunction
    Anxiety
    Glutamate regulation
    Panic Disorder (attacks)
    Stiffman Syndrome
GI disorder:
    Crohn’s
    Irritable bowel disease
    Ulcerative Colitis
Hormone dysfunction
    Cortisol dysfunction
    Premenstraul Syndrome
Hyperactivity
Insomnia
Lyme Disease
Migraines
    Abdominal
    Headache
    Atypical
Obsessive-Compulsive Disorder (OCD)
Organ system dysfunction
Phobias
Post-Traumatic stress disorder (PTSD)
Psychotic illness
Schizophrenia
Seasonal affective disorder
Social Anxiety disorder
Serotonin driven cardiac disease
Tension Headaches
Tourette’s Syndrome
Traumatic brain injury
Tricotillomania

MASTER SSRI-INDUCED SEROTONIN DEPLETION

  • Platelets contain 99% of whole blood serotonin. SSRI drugs may deplete 90% of platelet serotonin in three weeks. Eventually, lab assay may reveal serotonin concentrations that are undetectable.
  • Giving R&R with SSRI drugs may address SSRI-induced serotonin depletion
  • Low serotonin or dopamine is associated with suicidal ideation.

Learn to manage drug-induced serotonin depletion.

Warning: Suicidality and Antidepressant Drugs

See full prescribing information for complete boxed warning.
Increased risk of suicidal thinking and behavior in children, adolescents and young adults taking antidepressants for major depressive disorder (MDD) and other psychiatric disorders. Lexapro is no approved for use in pediatric patients less than 12 years of age (5.1).

 
Hinz Medical Foods Logo

Hinz Medical FoodsTM / NeuroResearch Centers, Inc.TM
1150 88th Ave. West – Duluth, MN +1-218-626-2220 | www.HinzMedicalFoods.com

HyposerotonergicTM
conditions
occur when
serotonin concentrations are
not enough, low, inadequate,
depleted, deficient, or
suboptimal on a modified
normal diet.TM

HypodopaminergicTM
conditions
occur when
dopamine concentrations are
not enough, low, inadequate,
depleted, deficient, or
suboptimal on a modified
normal diet.TM

HypoglutathionemiaTM
conditions
occur when
glutathione concentrations are
not enough, low, inadequate,
depleted, deficient, or
suboptimal on a modified
normal diet.TM

  • Giving only serotonin precursors can deplete dopamine and glutathione.TM

  • Giving only dopamine precursors can deplete serotonin and glutathione.TM

  • Giving only glutathione or glutathione precursors can deplete serotonin and dopamine.TM

The centrally acting monoamines (monoamines) are serotonin, dopamine, norephinephrine, and epinephrine.